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Artigo de periodico
Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids (2022)
Costa, Tiago Januário da ; Barros, Paula Rodrigues de ; Duarte, Diego A. ; Silva-Neto, Júlio Alves da; Hott, Sara Cristina ; Santos-Silva, Thamyris ; Costa-Neto, Claudio M. ; McCarthy, Cameron G. ; Jiménez-Altayó, Francesc ; Dantas, Ana Paula Villela ; Passaglia, Rita de Cassia Aleixo Tostes ; Akamine, Eliana Hiromi ; Gomes, Felipe Villela
Source: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FMRP, ICB
Subjects: FARMACOLOGIA, PROSTAGLANDINAS, RECEPTORES ADRENÉRGICOS, MENOPAUSA, FÁRMACOS (SISTEMA CARDIOVASCULAR), ARTÉRIAS CARÓTIDAS, VASOS SANGUÍNEOS, CAMUNDONGOS, VASODILATAÇÃO, MODELOS ANIMAIS DE DOENÇAS, ENVELHECIMENTO
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ABNT
COSTA, Tiago Januário da et al. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, v. 324, n. 4, p. H417-H429, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00495.2022. Acesso em: 30 abr. 2024.
APA
Costa, T. J. da, Barros, P. R. de, Duarte, D. A., Silva-Neto, J. A. da, Hott, S. C., Santos-Silva, T., et al. (2022). Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, 324( 4), H417-H429. doi:https://doi.org/10.1152/ajpheart.00495.2022
NLM
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2022 ; 324( 4): H417-H429.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Vancouver
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2022 ; 324( 4): H417-H429.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Artigo de periodico
NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes (2021)
Costa, Frederico Ribeiro Campos ; Leite, Jefferson Antonio; Rassi, Diane Meyre ; Silva, Josiane Fernandes da ; Oliveira, Jefferson Elias ; Guimarães, Jhefferson Barbosa ; Freitas, Maria Cristina Foss de ; Câmara, Niels Olsen Saraiva ; Pontillo, Alessandra ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Carlos, Daniela
Source: Cell Reports. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, DIABETES MELLITUS, DOENÇAS AUTOIMUNES, PÂNCREAS, CAMUNDONGOS, POLIMORFISMO, SISTEMA IMUNE, INTERLEUCINAS
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COSTA, Frederico Ribeiro Campos et al. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, v. 35, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2021.109176. Acesso em: 30 abr. 2024.
APA
Costa, F. R. C., Leite, J. A., Rassi, D. M., Silva, J. F. da, Oliveira, J. E., Guimarães, J. B., et al. (2021). NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, 35, 1-16. doi:10.1016/j.celrep.2021.109176
NLM
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Vancouver
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Artigo de periodico
Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence (2021)
Barros, Paula Rodrigues de ; Costa, Tiago Januário da ; Akamine, Eliana Hiromi ; Passaglia, Rita de Cassia Aleixo Tostes
Source: Frontiers in Aging. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, ENVELHECIMENTO, ENVELHECIMENTO CELULAR, DIFERENÇAS SEXUAIS (PSICOGÊNESE), MODELOS ANIMAIS DE DOENÇAS, ROEDORES, DOENÇAS CARDIOVASCULARES EM ANIMAL
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BARROS, Paula Rodrigues de et al. Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence. Frontiers in Aging, v. 2, p. 1-22, 2021Tradução . . Disponível em: https://doi.org/10.3389/fragi.2021.727604. Acesso em: 30 abr. 2024.
APA
Barros, P. R. de, Costa, T. J. da, Akamine, E. H., & Passaglia, R. de C. A. T. (2021). Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence. Frontiers in Aging, 2, 1-22. doi:10.3389/fragi.2021.727604
NLM
Barros PR de, Costa TJ da, Akamine EH, Passaglia R de CAT. Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence [Internet]. Frontiers in Aging. 2021 ; 2 1-22.[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fragi.2021.727604
Vancouver
Barros PR de, Costa TJ da, Akamine EH, Passaglia R de CAT. Vascular aging in rodent models: contrasting mechanisms driving the female and male vascular senescence [Internet]. Frontiers in Aging. 2021 ; 2 1-22.[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fragi.2021.727604
Artigo de periodico
The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway (2020)
Leite, Jefferson Antonio; Pessenda, Gabriela ; Gomes, Isabel Cristina Guerra ; Santana, Alynne Karen Mendonça de; Pereira, Camila André; Costa, Frederico Ribeiro Campos; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Faria, Ana Maria Caetano ; Almeida, Danilo Candido de ; Câmara, Niels Olsen Saraiva ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Cells. Unidades: FMRP, ICB
Assunto: IMUNOLOGIA
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ABNT
LEITE, Jefferson Antonio et al. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, v. 9, n. 4, p. 21 , 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9040959. Acesso em: 30 abr. 2024.
APA
Leite, J. A., Pessenda, G., Gomes, I. C. G., Santana, A. K. M. de, Pereira, C. A., Costa, F. R. C., et al. (2020). The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, 9( 4), 21 . doi:10.3390/cells9040959
NLM
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.3390/cells9040959
Vancouver
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.3390/cells9040959
Artigo de periodico
NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model (2020)
Sartori, Daniela Carlos ; Pérez, Malena Martínez ; Leite, Jefferson Antonio; Rocha, Fernanda Agostini; Martins, Larissa Marfori Sampaio; Pereira, Camila André; Silva, Thais Fernanda de Campos Fraga da ; Pucci, Taís Andrade; Ramos, Simone Gusmão ; Câmara, Niels Olsen Saraiva ; Bonato, Vânia Luiza Deperon ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB, FCFRP
Subjects: IMUNOLOGIA, LINFÓCITOS T, DIABETES MELLITUS, INFLAMAÇÃO, OBESIDADE, ADIPOSIDADE, CAMUNDONGOS, RESISTÊNCIA À INSULINA, HIPERGLICEMIA, LINFONODOS DE ANIMAL, EXPRESSÃO GÊNICA, BACTÉRIAS GRAM-NEGATIVAS, BACTEROIDES
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ABNT
SARTORI, Daniela Carlos et al. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, v. 11, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01265. Acesso em: 30 abr. 2024.
APA
Sartori, D. C., Pérez, M. M., Leite, J. A., Rocha, F. A., Martins, L. M. S., Pereira, C. A., et al. (2020). NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, 11, 16 . doi:10.3389/fimmu.2020.01265
NLM
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Vancouver
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Artigo de periodico
Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry (2019)
Filgueira, Fernando Paranaiba; Lobato, Núbia de Souza; Nascimento, Denise L.; Ceravolo, Graziela Scalianti; Giachini, Fernanda R. C.; Lima, Victor V.; Dantas, Ana Paula; Fortes, Zuleica Bruno; Webb, R. Clinton; Tostes, Rita de Cássia Aleixo ; Carvalho, Maria Helena Catelli de
Source: Steroids. Unidades: ICB, FMRP
Subjects: ESTRADIOL, ARTÉRIAS, VASODILATAÇÃO
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FILGUEIRA, Fernando Paranaiba et al. Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry. Steroids, v. 141, p. 46-54, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.steroids.2018.11.006. Acesso em: 30 abr. 2024.
APA
Filgueira, F. P., Lobato, N. de S., Nascimento, D. L., Ceravolo, G. S., Giachini, F. R. C., Lima, V. V., et al. (2019). Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry. Steroids, 141, 46-54. doi:10.1016/j.steroids.2018.11.006
NLM
Filgueira FP, Lobato N de S, Nascimento DL, Ceravolo GS, Giachini FRC, Lima VV, Dantas AP, Fortes ZB, Webb RC, Tostes R de CA, Carvalho MHC de. Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry [Internet]. Steroids. 2019 ; 141 46-54.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.steroids.2018.11.006
Vancouver
Filgueira FP, Lobato N de S, Nascimento DL, Ceravolo GS, Giachini FRC, Lima VV, Dantas AP, Fortes ZB, Webb RC, Tostes R de CA, Carvalho MHC de. Equilin displays similar endothelium-independent vasodilator potential to 17β-estradiol regardless of lower potential to inhibit calcium entry [Internet]. Steroids. 2019 ; 141 46-54.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.steroids.2018.11.006
Artigo de periodico
Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 (2019)
Costa, Tiago Januário ; Altayó, Francesc Jiménez ; Pereira, Cinthya Echem de Souza; Akamine, Eliana Hiromi ; Tostes, Rita de Cássia Aleixo ; Vila, Elisabet ; Dantas, Ana Paula ; Carvalho, Maria Helena Catelli de
Source: Cells. Unidades: ICB, FMRP
Subjects: FAMACOLOGIA, ESTRÓGENOS, ARTÉRIAS CARÓTIDAS, EICOSANOIDES, ENSAIO CLÍNICO, ESTRADIOL, OVARIECTOMIA, CAMUNDONGOS
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ABNT
COSTA, Tiago Januário et al. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36. Cells, v. 8, n. 10, p. 17 , 2019Tradução . . Disponível em: https://doi.org/10.3390/cells8101217. Acesso em: 30 abr. 2024.
APA
Costa, T. J., Altayó, F. J., Pereira, C. E. de S., Akamine, E. H., Tostes, R. de C. A., Vila, E., et al. (2019). Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36. Cells, 8( 10), 17 . doi:10.3390/cells8101217
NLM
Costa TJ, Altayó FJ, Pereira CE de S, Akamine EH, Tostes R de CA, Vila E, Dantas AP, Carvalho MHC de. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 [Internet]. Cells. 2019 ; 8( 10): 17 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.3390/cells8101217
Vancouver
Costa TJ, Altayó FJ, Pereira CE de S, Akamine EH, Tostes R de CA, Vila E, Dantas AP, Carvalho MHC de. Late onset of estrogen therapy impairs carotid function of senescent females in association with altered prostanoid balance and upregulation of the variant ERα36 [Internet]. Cells. 2019 ; 8( 10): 17 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.3390/cells8101217
Artigo de periodico
Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model (2017)
Carlos, Daniela ; Costa, Frederico Ribeiro Campos ; Pereira, Camila André; Rocha, Fernanda Agostini; Yaochite, Juliana Navarro Ueda; Oliveira, Gabriela G.; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Câmara, Niels Olsen Saraiva ; Ryffel, Bernhard; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB
Subjects: CITOCINAS, DIABETES MELLITUS, DNA MITOCONDRIAL
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CARLOS, Daniela et al. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00164. Acesso em: 30 abr. 2024.
APA
Carlos, D., Costa, F. R. C., Pereira, C. A., Rocha, F. A., Yaochite, J. N. U., Oliveira, G. G., et al. (2017). Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00164
NLM
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Vancouver
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Artigo de periodico
H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction (2016)
Costa, Rafael M.; Filgueira, Fernando P.; Passaglia, Rita de Cássia Aleixo Tostes; Carvalho, Maria Helena C.; Akamine, Eliana Hiromi ; Lobato, Nubia S.
Source: Vascular Pharmacology. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
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COSTA, Rafael M. et al. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, v. 84, p. 28-37, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.008. Acesso em: 30 abr. 2024.
APA
Costa, R. M., Filgueira, F. P., Passaglia, R. de C. A. T., Carvalho, M. H. C., Akamine, E. H., & Lobato, N. S. (2016). H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction. Vascular Pharmacology, 84, 28-37. doi:10.1016/j.vph.2016.05.008
NLM
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Vancouver
Costa RM, Filgueira FP, Passaglia R de CAT, Carvalho MHC, Akamine EH, Lobato NS. H2O2 generated from mitochondrial electron transport chain in thoracic perivascular adipose tissue is crucial for modulation of vascular smooth muscle contraction [Internet]. Vascular Pharmacology. 2016 ; 84 28-37.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.vph.2016.05.008
Artigo de periodico
Reactive oxygen species: players in the cardiovascular effects of testosterone (2016)
Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva; Carvalho, Maria Helena Catelli de; Reckelhoff, Jane F.
Source: American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. Unidades: FMRP, ICB
Subjects: DOENÇAS CARDIOVASCULARES, SISTEMA CARDIOVASCULAR, TERAPIA DA REPOSIÇÃO HORMONAL, ESTRESSE OXIDATIVO, TESTOSTERONA
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ABNT
TOSTES, Rita de Cássia Aleixo et al. Reactive oxygen species: players in the cardiovascular effects of testosterone. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, v. 310, n. 1, p. R1-R14, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00392.2014. Acesso em: 30 abr. 2024.
APA
Tostes, R. de C. A., Carneiro, F. S., Carvalho, M. H. C. de, & Reckelhoff, J. F. (2016). Reactive oxygen species: players in the cardiovascular effects of testosterone. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, 310( 1), R1-R14. doi:10.1152/ajpregu.00392.2014
NLM
Tostes R de CA, Carneiro FS, Carvalho MHC de, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects of testosterone [Internet]. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Vancouver
Tostes R de CA, Carneiro FS, Carvalho MHC de, Reckelhoff JF. Reactive oxygen species: players in the cardiovascular effects of testosterone [Internet]. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology. 2016 ; 310( 1): R1-R14.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpregu.00392.2014
Artigo de periodico
NLRP3 inflammasome mediates aldosterone-induced vascular damage (2016)
Bruder-Nascimento, Thiago; Ferreira, Nathanne S.; Zanotto, Camila Z.; Ramalho, Fernanda; Pequeno, Isabela O.; Olivon, Vania C.; Neves, Karla B.; Alves-Lopes, Rheure; Campos, Eduardo; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens; Carlos, Daniela; Mestriner, Fabiola Leslie Antunes Cardoso; Prado, Douglas; Pereira, Felipe V.; Braga, Tarcio; Luiz, Joao Paulo M.; Cau, Stefany B.; Elias, Paula C.; Moreira, Ayrton Custódio; Câmara, Niels Olsen Saraiva ; Zamboni, Dario Simões; Alves Filho, José Carlos Farias; Tostes, Rita de Cássia Aleixo
Source: Circulation. Unidades: FMRP, ICB
Subjects: ALDOSTERONA, IMUNOLOGIA, INFLAMAÇÃO, DOENÇAS VASCULARES, INTERLEUCINAS
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ABNT
BRUDER-NASCIMENTO, Thiago et al. NLRP3 inflammasome mediates aldosterone-induced vascular damage. Circulation, v. 134, n. 23, p. 1866-1880, 2016Tradução . . Disponível em: https://doi.org/10.1161/circulationaha.116.024369. Acesso em: 30 abr. 2024.
APA
Bruder-Nascimento, T., Ferreira, N. S., Zanotto, C. Z., Ramalho, F., Pequeno, I. O., Olivon, V. C., et al. (2016). NLRP3 inflammasome mediates aldosterone-induced vascular damage. Circulation, 134( 23), 1866-1880. doi:10.1161/circulationaha.116.024369
NLM
Bruder-Nascimento T, Ferreira NS, Zanotto CZ, Ramalho F, Pequeno IO, Olivon VC, Neves KB, Alves-Lopes R, Campos E, Silva CAA da, Fazan Júnior R, Carlos D, Mestriner FLAC, Prado D, Pereira FV, Braga T, Luiz JPM, Cau SB, Elias PC, Moreira AC, Câmara NOS, Zamboni DS, Alves Filho JCF, Tostes R de CA. NLRP3 inflammasome mediates aldosterone-induced vascular damage [Internet]. Circulation. 2016 ; 134( 23): 1866-1880.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1161/circulationaha.116.024369
Vancouver
Bruder-Nascimento T, Ferreira NS, Zanotto CZ, Ramalho F, Pequeno IO, Olivon VC, Neves KB, Alves-Lopes R, Campos E, Silva CAA da, Fazan Júnior R, Carlos D, Mestriner FLAC, Prado D, Pereira FV, Braga T, Luiz JPM, Cau SB, Elias PC, Moreira AC, Câmara NOS, Zamboni DS, Alves Filho JCF, Tostes R de CA. NLRP3 inflammasome mediates aldosterone-induced vascular damage [Internet]. Circulation. 2016 ; 134( 23): 1866-1880.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1161/circulationaha.116.024369
Artigo de periodico
Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats (2015)
Bomfim, G. F.; Echem, Cinthya; Martins, C. B.; Costa, T. J.; Sartoretto, Simone Marcieli; Santos, R. A. dos; Oliveira, M. A.; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo; Webb, R. C.; Carvalho, Maria Helena Catelli de
Source: Life Sciences. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, INFLAMAÇÃO, HIPERTENSÃO, RATOS
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ABNT
BOMFIM, G. F. et al. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, v. 122, p. 1-7, 2015Tradução . . Disponível em: http://lattes.cnpq.br/5055165961832291. Acesso em: 30 abr. 2024.
APA
Bomfim, G. F., Echem, C., Martins, C. B., Costa, T. J., Sartoretto, S. M., Santos, R. A. dos, et al. (2015). Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats. Life Sciences, 122, 1-7. doi:10.1016/j.lfs.2014.12.001
NLM
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 abr. 30 ] Available from: http://lattes.cnpq.br/5055165961832291
Vancouver
Bomfim GF, Echem C, Martins CB, Costa TJ, Sartoretto SM, Santos RA dos, Oliveira MA, Akamine EH, Fortes ZB, Tostes R de CA, Webb RC, Carvalho MHC de. Toll-like receptor 4 inhibition reduces vascular inflammation in spontaneously hypertensive rats [Internet]. Life Sciences. 2015 ; 122 1-7.[citado 2024 abr. 30 ] Available from: http://lattes.cnpq.br/5055165961832291
Artigo de periodico
Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells (2015)
Costa, Tiago Januário da; Ceravolo, Graziela Scalianti; Santos, Rosangela Aparecida dos; Oliveira, Maria Aparecida de; Araújo, Priscila Xavier de; Giaquinto, Luciana Rigueiral; Tostes, Rita de Cássia Aleixo; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Dantas, Ana Paula; Carvalho, Maria Helena Catelli de
Source: American Journal of Physiology: Heart and Circulatory Physiology. Unidades: ICB, FMRP
Subjects: FARMACOLOGIA, TESTOSTERONA, ESTROGENOS, AORTA DE ANIMAL, CÉLULAS ENDOTELIAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells. American Journal of Physiology: Heart and Circulatory Physiology, v. 308, n. 7, p. H723-H732, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00681.2014. Acesso em: 30 abr. 2024.
APA
Costa, T. J. da, Ceravolo, G. S., Santos, R. A. dos, Oliveira, M. A. de, Araújo, P. X. de, Giaquinto, L. R., et al. (2015). Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells. American Journal of Physiology: Heart and Circulatory Physiology, 308( 7), H723-H732. doi:10.1152/ajpheart.00681.2014
NLM
Costa TJ da, Ceravolo GS, Santos RA dos, Oliveira MA de, Araújo PX de, Giaquinto LR, Tostes R de CA, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC de. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2015 ; 308( 7): H723-H732.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpheart.00681.2014
Vancouver
Costa TJ da, Ceravolo GS, Santos RA dos, Oliveira MA de, Araújo PX de, Giaquinto LR, Tostes R de CA, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC de. Association of testosterone with estrogen abolishes the beneficial effects of estrogen treatment by increasing ROS generation in aorta endothelial cells [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2015 ; 308( 7): H723-H732.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpheart.00681.2014
Artigo de periodico
Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms (2015)
Chignalia, Andreia Z; Oliveira, Maria Aparecida; Debbas, Victor; Dull, Randal O; Laurindo, Francisco R. M; Touyz, Rhian M; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidades: FMRP, FM, ICB
Subjects: ANDRÓGENOS (FARMACOLOGIA), INIBIDORES DE ENZIMAS, LEUCÓCITOS (METABOLISMO), TRANSDUÇÃO DE SINAL CELULAR, TESTOSTERONA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHIGNALIA, Andreia Z et al. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, v. 129, n. 1, p. 39-48, 2015Tradução . . Disponível em: https://doi.org/10.1042/CS20140548. Acesso em: 30 abr. 2024.
APA
Chignalia, A. Z., Oliveira, M. A., Debbas, V., Dull, R. O., Laurindo, F. R. M., Touyz, R. M., et al. (2015). Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, 129( 1), 39-48. doi:10.1042/CS20140548
NLM
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1042/CS20140548
Vancouver
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1042/CS20140548
Artigo de periodico
An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies (2014)
Ceravolo, Graziela S.; Montezano, Augusto C.; Jordão, Maria T.; Akamine, Eliana Hiromi ; Costa, Tiago Januário da; Takano, Ana Paula Cremasco ; Fernandes, Denise C.; Barreto-Chaves, Maria Luiza Morais ; Laurindo, Francisco R.; Tostes, Rita de Cássia; Fortes, Zuleica Bruno; Chopard, Renato Paulo; Touyz, Rhian M.; Carvalho, Maria Helena Catelli de
Source: PLOS ONE. Unidades: ICB, FMRP
Subjects: ANATOMIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CERAVOLO, Graziela S. et al. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, v. 9, n. 11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0111117. Acesso em: 30 abr. 2024.
APA
Ceravolo, G. S., Montezano, A. C., Jordão, M. T., Akamine, E. H., Costa, T. J. da, Takano, A. P. C., et al. (2014). An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, 9( 11). doi:10.1371/journal.pone.0111117
NLM
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 abr. 30 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Vancouver
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 abr. 30 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Artigo de periodico
Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats (2013)
Sartoretto, Simone Marcieli; Santos-Eichler, Rosângela; Tostes, Rita de Cássia Aleixo; Carvalho, Maria Helena Catelli de; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno
Source: Journal of Diabetes. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SARTORETTO, Simone Marcieli et al. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, v. 5, n. 2, p. 197-206, 2013Tradução . . Acesso em: 30 abr. 2024.
APA
Sartoretto, S. M., Santos-Eichler, R., Tostes, R. de C. A., Carvalho, M. H. C. de, Akamine, E. H., & Fortes, Z. B. (2013). Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, 5( 2), 197-206.
NLM
Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 abr. 30 ]
Vancouver
Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 abr. 30 ]
Artigo de periodico
Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats (2013)
Ceravolo, Graziela Scalianti; Filgueira, Fernando Paranaiba; Costa, Tiago Januário da; Lobato, Núbia de Souza; Chignalia, Andréia Zago; Araujo, Priscila Xavier de; Tostes, Rita de Cassia; Dantas, Ana P.; Fortes, Zuleica Bruno; Carvalho, Maria Helena Catelli de
Source: Steroids. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CERAVOLO, Graziela Scalianti et al. Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats. Steroids, v. 78, n. 3, p. 341-346, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.steroids.2012.11.018. Acesso em: 30 abr. 2024.
APA
Ceravolo, G. S., Filgueira, F. P., Costa, T. J. da, Lobato, N. de S., Chignalia, A. Z., Araujo, P. X. de, et al. (2013). Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats. Steroids, 78( 3), 341-346. doi:10.1016/j.steroids.2012.11.018
NLM
Ceravolo GS, Filgueira FP, Costa TJ da, Lobato N de S, Chignalia AZ, Araujo PX de, Tostes R de C, Dantas AP, Fortes ZB, Carvalho MHC de. Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats [Internet]. Steroids. 2013 ; 78( 3): 341-346.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.steroids.2012.11.018
Vancouver
Ceravolo GS, Filgueira FP, Costa TJ da, Lobato N de S, Chignalia AZ, Araujo PX de, Tostes R de C, Dantas AP, Fortes ZB, Carvalho MHC de. Conjugated equine estrogen treatment corrected the exacerbated aorta oxidative stress in ovariectomized spontaneously hypertensive rats [Internet]. Steroids. 2013 ; 78( 3): 341-346.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.steroids.2012.11.018
Artigo de periodico
Reduced endothelium-dependent relaxation to anandamide in mesenteric arteries from young obese Zucker rats (2013)
Lobato, Nubia S.; Filgueira, Fernando P.; Prakash, Roshini; Giachini, Fernanda R.; Ergul, Adviye; Carvalho, Maria Helena Catelli de; Webb, R. Clinton; Tostes, Rita de Cassia; Fortes, Zuleica Bruno
Source: PLOS ONE. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOBATO, Nubia S. et al. Reduced endothelium-dependent relaxation to anandamide in mesenteric arteries from young obese Zucker rats. PLOS ONE, v. 8, n. 5, p. 1-11, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0063449. Acesso em: 30 abr. 2024.
APA
Lobato, N. S., Filgueira, F. P., Prakash, R., Giachini, F. R., Ergul, A., Carvalho, M. H. C. de, et al. (2013). Reduced endothelium-dependent relaxation to anandamide in mesenteric arteries from young obese Zucker rats. PLOS ONE, 8( 5), 1-11. doi:10.1371/journal.pone.0063449
NLM
Lobato NS, Filgueira FP, Prakash R, Giachini FR, Ergul A, Carvalho MHC de, Webb RC, Tostes R de C, Fortes ZB. Reduced endothelium-dependent relaxation to anandamide in mesenteric arteries from young obese Zucker rats [Internet]. PLOS ONE. 2013 ; 8( 5): 1-11.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1371/journal.pone.0063449
Vancouver
Lobato NS, Filgueira FP, Prakash R, Giachini FR, Ergul A, Carvalho MHC de, Webb RC, Tostes R de C, Fortes ZB. Reduced endothelium-dependent relaxation to anandamide in mesenteric arteries from young obese Zucker rats [Internet]. PLOS ONE. 2013 ; 8( 5): 1-11.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1371/journal.pone.0063449
Artigo de periodico
Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism (2013)
Carrillo-Sepúlveda, Maria Alícia; Ceravolo, Graziela S.; Furstenau, Cristina R.; Monteiro, Priscilla de Souza; Bruno-Fortes, Zuleica; Carvalho, Maria Helena Catelli de; Laurindo, Francisco R.; Tostes, Rita de Cássia; Webb, R. Clinton; Barreto-Chaves, Maria Luiza Morais
Source: PLOS ONE. Unidades: ICB, FMRP
Subjects: ANATOMIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARRILLO-SEPÚLVEDA, Maria Alícia et al. Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism. PLOS ONE, v. 8, n. 4, p. 1-9, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0061982. Acesso em: 30 abr. 2024.
APA
Carrillo-Sepúlveda, M. A., Ceravolo, G. S., Furstenau, C. R., Monteiro, P. de S., Bruno-Fortes, Z., Carvalho, M. H. C. de, et al. (2013). Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism. PLOS ONE, 8( 4), 1-9. doi:10.1371/journal.pone.0061982
NLM
Carrillo-Sepúlveda MA, Ceravolo GS, Furstenau CR, Monteiro P de S, Bruno-Fortes Z, Carvalho MHC de, Laurindo FR, Tostes R de C, Webb RC, Barreto-Chaves MLM. Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism [Internet]. PLOS ONE. 2013 ;8( 4): 1-9.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1371/journal.pone.0061982
Vancouver
Carrillo-Sepúlveda MA, Ceravolo GS, Furstenau CR, Monteiro P de S, Bruno-Fortes Z, Carvalho MHC de, Laurindo FR, Tostes R de C, Webb RC, Barreto-Chaves MLM. Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism [Internet]. PLOS ONE. 2013 ;8( 4): 1-9.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1371/journal.pone.0061982
Artigo de periodico
Mechanisms of endothelial dysfunction in obesity-associated hypertension (2012)
Lobato, Nubia de Souza; Filgueira, Fernando Paranaiba; Akamine, Eliana Hiromi ; Passaglia, Rita de Cassia Aleixo Tostes; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno
Source: Brazilian Journal of Medical and Biological Research. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOBATO, Nubia de Souza et al. Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, v. 45, n. 5, p. 392-400, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2012007500058. Acesso em: 30 abr. 2024.
APA
Lobato, N. de S., Filgueira, F. P., Akamine, E. H., Passaglia, R. de C. A. T., Carvalho, M. H. C. de, & Fortes, Z. B. (2012). Mechanisms of endothelial dysfunction in obesity-associated hypertension. Brazilian Journal of Medical and Biological Research, 45( 5), 392-400. doi:10.1590/S0100-879X2012007500058
NLM
Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058
Vancouver
Lobato N de S, Filgueira FP, Akamine EH, Passaglia R de CAT, Carvalho MHC de, Fortes ZB. Mechanisms of endothelial dysfunction in obesity-associated hypertension [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 5): 392-400.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1590/S0100-879X2012007500058

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